Search results for "Matrix Metalloproteinase 13"

showing 9 items of 9 documents

Molecular interactions between human cartilaginous endplates and nucleus pulposus cells: a preliminary investigation.

2014

Study Design. Conditioned media (CM) of cartilaginous endplates (CEPs) of intervertebral discs were analyzed in a bioassay with regard to their influence on matrix turnover and inflammatory factors on nucleus pulposus (NP) cells of the same patient. CEP tissue underwent further histological and ultrastructural analysis. Objective. To identify possible interactions between the CEP and the disc via molecular factors that may influence disc matrix degradation and to determine degenerative changes of CEP tissue. Summary of Background Data. Impaired endplate perme-ability due to degeneration and calcification is considered to be a key contributor to disc degeneration. An upregulation of metallop…

AdultMalePathologymedicine.medical_specialtyCellIntervertebral Disc DegenerationMatrix metalloproteinaseMatrix (biology)Proinflammatory cytokineDownregulation and upregulationMatrix Metalloproteinase 13MedicineHumansOrthopedics and Sports MedicineAggrecansIntervertebral DiscAggrecanCells CulturedAgedbusiness.industryInterleukin-6Interleukin-8Middle AgedCell biologyTissue Degenerationmedicine.anatomical_structureCartilageTumor necrosis factor alphaFemaleMatrix Metalloproteinase 3Neurology (clinical)businessSpine
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Characterization of collagenase 3 (matrix metalloproteinase 13) messenger RNA expression in the synovial membrane and synovial fibroblasts of patient…

1999

Objective To study the localization and cell type–specific expression of collagenase 3 messenger RNA (mRNA) in the synovial membrane, its regulation in primary synovial fibroblasts, and the correlation with systemic markers of inflammation and radiographic damage in rheumatoid arthritis (RA). Methods The expression of collagenase 3 mRNA was characterized by Northern blot analysis, reverse transcriptase–polymerase chain reaction, and in situ hybridization. Immunohistochemical detection of cell type–specific antigens was used in combination with in situ hybridization of collagenase 3 mRNA to characterize the cellular origin of collagenase 3 mRNA expression. Results Collagenase 3 mRNA was dete…

AdultMalePathologymedicine.medical_specialtyPhosphodiesterase InhibitorsImmunologyIn situ hybridizationBiologyArthritis RheumatoidRheumatology1-Methyl-3-isobutylxanthineMatrix Metalloproteinase 13Cyclic AMPmedicineHumansImmunology and AllergyPharmacology (medical)CollagenasesRNA MessengerNorthern blotFibroblastCells CulturedIn Situ HybridizationAgedAged 80 and overMessenger RNAColforsinSynovial MembraneFibroblastsMiddle AgedMolecular biologyEnzyme ActivationRadiographymedicine.anatomical_structureBucladesineGene Expression RegulationCell cultureCollagenaseInterstitial collagenaseFemaleSynovial membraneAdenylyl Cyclasesmedicine.drugArthritis & Rheumatism
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Genetic abrogation of the fibronectin-α5β1 integrin interaction in articular cartilage aggravates osteoarthritis in mice.

2018

The balance between synthesis and degradation of the cartilage extracellular matrix is severely altered in osteoarthritis, where degradation predominates. One reason for this imbalance is believed to be due to the ligation of the α5β1 integrin, the classic fibronectin (FN) receptor, with soluble FN fragments instead of insoluble FN fibrils, which induces matrix metalloproteinase (MMP) expression. Our objective was to determine whether the lack of α5β1-FN binding influences cartilage morphogenesis in vivo and whether non-ligated α5β1 protects or aggravates the course of osteoarthritis in mice. We engineered mice (Col2a-Cre;Fn1RGE/fl), whose chondrocytes express an α5β1 binding-deficient FN, …

Cartilage ArticularMale0301 basic medicineIntegrinsKnee JointGlycobiologylcsh:MedicineCartilage morphogenesisOsteoarthritisMatrix metalloproteinaseBiochemistryExtracellular matrixMice0302 clinical medicineAnimal CellsMedicine and Health Scienceslcsh:ScienceConnective Tissue CellsStainingMultidisciplinarybiologyChemistryExtracellular MatrixCell biologymedicine.anatomical_structureConnective TissueProteoglycansMatrix Metalloproteinase 3AnatomyCellular Structures and OrganellesCellular TypesResearch ArticleIntegrin alpha5beta1Signal TransductionIntegrinMice TransgenicResearch and Analysis Methods03 medical and health sciencesChondrocytesPhysical Conditioning AnimalMatrix Metalloproteinase 13OsteoarthritisCell AdhesionmedicineAnimalsHumansRegenerationCytoplasmic Staining030203 arthritis & rheumatologyCartilagelcsh:RBiology and Life SciencesProteinsCell Biologymedicine.diseaseFibronectinsFibronectinDisease Models AnimalBiological TissueCartilage030104 developmental biologyProteoglycanSpecimen Preparation and Treatmentbiology.proteinSafranin Staininglcsh:QCollagensArticular CartilagePLoS ONE
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Induction of collagenase-3 (MMP-13) expression in human skin fibroblasts by three-dimensional collagen is mediated by p38 mitogen-activated protein k…

1999

Collagenase-3 (matrix metalloproteinase-13, MMP-13) is a recently identified human MMP with an exceptionally wide substrate specificity and restricted tissue-specific expression. Here we show that MMP-13 expression is induced in normal human skin fibroblasts cultured within three-dimensional collagen gel resulting in production and proteolytic activation of MMP-13. Induction of MMP-13 mRNAs by collagen gel was potently inhibited by blocking antibodies against alpha1 and alpha2 integrin subunits and augmented by activating antibody against beta1 integrin subunit, indicating that both alpha1 beta1 and alpha2 beta1 integrins mediate the MMP-13-inducing cellular signal generated by three-dimens…

MAPK/ERK pathwayIntegrinsReceptors CollagenSB 203580IntegrinDown-RegulationBiologyBiochemistryp38 Mitogen-Activated Protein KinasesCollagen receptorIntegrin alpha1beta1chemistry.chemical_compoundTransforming Growth Factor betaMatrix Metalloproteinase 13medicineHumansCollagenasesProtein kinase AMolecular BiologyDNA PrimersSkinBase SequenceKinaseTumor Necrosis Factor-alphaCell BiologyFibroblastsProtein-Tyrosine KinasesMolecular biologyEnzyme ActivationchemistryCalcium-Calmodulin-Dependent Protein KinasesCollagenasebiology.proteinCollagenMitogen-Activated Protein KinasesTyrosine kinasemedicine.drugInterleukin-1The Journal of biological chemistry
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Conditioned Media from Adipose-Tissue-Derived Mesenchymal Stem Cells Downregulate Degradative Mediators Induced by Interleukin-1β in Osteoarthritic C…

2013

Osteoarthritis (OA) is the most frequent joint disorder and an important cause of disability. Recent studies have shown the potential of adipose-tissue-derived mesenchymal stem cells (AD-MSC) for cartilage repair. We have investigated whether conditioned medium from AD-MSC (CM) may regulate in OA chondrocytes a number of key mediators involved in cartilage degeneration. CM enhanced type II collagen expression in OA chondrocytes while decreasing matrix metalloproteinase (MMP) activity in cell supernatants as well as the levels of MMP-3 and MMP-13 proteins and mRNA in OA chondrocytes stimulated with interleukin- (IL-) 1β. In addition, CM increased IL-10 levels and counteracted the stimulating…

MaleArticle Subjectmedicine.medical_treatmentImmunologyInterleukin-1betaType II collagenAdipose tissueDown-RegulationNitric OxideChondrocytesMatrix Metalloproteinase 13Osteoarthritislcsh:PathologymedicineHumansProstaglandin E2Interleukin 6Collagen Type IICells CulturedAgedbiologyChemistryInterleukin-6Tumor Necrosis Factor-alphaMesenchymal stem cellNF-kappa BInterleukinMesenchymal Stem CellsCell BiologyMiddle AgedCell biologyAdipose TissueCulture Media ConditionedImmunologybiology.proteinTumor necrosis factor alphaFemaleMatrix Metalloproteinase 3Inflammation Mediatorslcsh:RB1-214Prostaglandin Emedicine.drugResearch Article
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High mobility group box 1 potentiates the pro-inflammatory effects of interleukin-1β in osteoarthritic synoviocytes

2010

Introduction High mobility group box 1 (HMGB1) is released by necrotic cells or secreted in response to inflammatory stimuli. Extracellular HMGB1 may act as a pro-inflammatory cytokine in rheumatoid arthritis. We have recently reported that HMGB1 is released by osteoarthritic synoviocytes after activation with interleukin-1beta (IL-1β) The present study investigated the role of HMGB1 in synovial inflammation in osteoarthritis (OA). Methods HMGB1 was determined in human synovium using immunohistochemistry, comparing normal to OA. OA synoviocytes were incubated with HMGB1 at 15 or 25 ng/ml in the absence or presence of IL-1β (10 ng/ml). Gene expression was analyzed by quantitative PCR and pro…

MaleChemokineMAP Kinase Signaling Systemmedicine.medical_treatmentInterleukin-1betaImmunologyInflammationchemical and pharmacologic phenomenaCCL2HMGB1p38 Mitogen-Activated Protein KinasesRheumatologySynovitisMatrix Metalloproteinase 13HumansMedicineImmunology and AllergyRNA MessengerHMGB1 ProteinExtracellular Signal-Regulated MAP KinasesCells CulturedAgedbiologybusiness.industrySynovial MembraneNF-kappa BOsteoarthritis Kneemedicine.diseaseImmunohistochemistryMolecular biologyCCL20Cytokinemedicine.anatomical_structurebiology.proteinFemaleMatrix Metalloproteinase 3Matrix Metalloproteinase 1Synovial membranemedicine.symptombusinessProto-Oncogene Proteins c-aktResearch ArticleArthritis Research & Therapy
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Collagenase-3 (MMP-13) Enhances Remodeling of Three-Dimensional Collagen and Promotes Survival of Human Skin Fibroblasts

2006

Collagenase-3 (MMP-13) is a matrix metalloproteinase capable of cleaving a multitude of extracellular matrix proteins in addition to fibrillar collagens. Human MMP-13 is expressed by fibroblasts in chronic cutaneous ulcers, but not in normally healing adult skin wounds. However, MMP-13 is produced by fibroblasts in adult gingival and in fetal skin wounds characterized by rapid collagen remodeling and scarless healing. Here, we have examined the role of human MMP-13 in remodeling of three-dimensional (3D) collagenous matrix by primary adult human skin fibroblasts. The high level of human MMP-13 expression by fibroblasts achieved by adenoviral gene delivery resulted in potent enhancement of r…

Pathologymedicine.medical_specialtyCell SurvivalHuman skinDermatologyMatrix Metalloproteinase InhibitorsMatrix metalloproteinaseBiologyBiochemistryFilamentous actinAdenoviridaeDermal fibroblastExtracellular matrixMatrix Metalloproteinase 13medicineHumansFibroblastMolecular BiologyCells CulturedCell ProliferationWound HealingTissue Inhibitor of Metalloproteinase-1integumentary systemCell BiologyFibroblastsActinsCell biologyEnzyme ActivationCollagen type I alpha 1medicine.anatomical_structureCollagenaseCollagenmedicine.drugJournal of Investigative Dermatology
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MMP-13 stimulates osteoclast differentiation and activation in tumour breast bone metastases

2011

INTRODUCTION: The increased bone degradation in osteolytic metastases depends on stimulation of mature osteoclasts and on continuous differentiation of new pre-osteoclasts. Metalloproteinases (MMP)-13 is expressed in a broad range of primary malignant tumours and it is emerging as a novel biomarker. Recent data suggest a direct role of MMP-13 in dissolving bone matrix complementing the activity of MMP-9 and other enzymes. Tumour-microenvironment interactions alter gene expression in malignant breast tumour cells promoting osteolytic bone metastasis. Gene expression profiles revealed that MMP-13 was among the up-regulated genes in tumour-bone interface and its abrogation reduced bone erosion…

Pathologymedicine.medical_specialtyCellular differentiationGalectin 3Mice NudeOsteoclastsBone NeoplasmsBreast NeoplasmsMatrix metalloproteinaseAdenocarcinomaExtracellular matrixMiceOsteoclastCell Line TumorMatrix Metalloproteinase 13medicineAnimalsHumansProtein PrecursorsOSTEOCLASTMedicine(all)MMP13 ; OSTEOCLAST; BREAST TUMORChemistryMMP13Bone metastasisCell Differentiationmedicine.diseaseXenograft Model Antitumor AssaysResorptionExtracellular Matrixmedicine.anatomical_structureCellular MicroenvironmentMatrix Metalloproteinase 9Galectin-3Cancer researchCytokinesFemaleBone marrowBREAST TUMORResearch ArticleBreast Cancer Research : BCR
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Immunohistochemical and transcriptional expression of matrix metalloproteinases in full-term human umbilical cord and Human Umbilical Vein Endothelia…

2010

Matrix metalloproteinases (MMPs) are extracellular zinc-dependent endopeptidases involved in the degradation and remodelling of extracellular matrix in physiological and pathological processes. MMPs also have a role on cell proliferation, migration, differentiation, angiogenesis and apoptosis. Umbilical cord is a special organ subjected to many changes during pre-natal life and whose cells can maintain a certain degree of plasticity also in post-natal period; for example recently they have been used as a source of stem cells. In this work we investigated the expression of MMPs in human umbilical cord and Human Umbilical Vein Endothelial Cells (HUVEC) though immunohistochemistry, RT-PCR and …

Settore BIO/17 - IstologiaUmbilical VeinsHistologyPhysiologyAngiogenesisBiologyMatrix metalloproteinaseUmbilical cordGene Expression Regulation EnzymologicUmbilical veinUmbilical CordExtracellular matrixMatrix Metalloproteinase 10Matrix Metalloproteinase 13ExtracellularmedicineHumansCells CulturedReverse Transcriptase Polymerase Chain ReactionEndothelial CellsCell BiologyGeneral MedicineMatrix metalloproteinases human umbilical cord HUVEC.ImmunohistochemistryMatrix MetalloproteinasesCord liningCell biologyMatrix Metalloproteinase 8medicine.anatomical_structureMatrix Metalloproteinase 9ImmunologyMatrix Metalloproteinase 2Matrix Metalloproteinase 3Stem cellJournal of Molecular Histology
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